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Kwerk, you should sign up for this "health drink" thread.
I don't see why not, many a time I have ran home through the streets of London at 2am smashed and been astonished by my speed, stamina and prodigous level of coolness while doing so.
I find it weird that not only do you manage to find a beer that is made in small batches in a microbrewery in the US and is shipped to a few select states, but you then drink it before / at the gym in the UK.
LOLKwerk.
Kwerk lives in the US.
That's because Kwerk is a berk and usually posts sensationalist rubbish.I love how most of you laugh at Kwerk
That's because Kwerk is a berk and usually posts sensationalist rubbish.
It's quite simple really. Alcohol thins your blood so your heart must be able to pump it round faster. More blood = more fasterness
The thing is, I'm sure alcohol is better than nothing.
I bet a proper pre-workout supplement such as Jack3D is better than alcohol though.
Next they'll be giving us scientific proof that a coffee and banana before a session will he--... WAIT A SECOND!
Ibuprofen or asprin would be a better choice though, Its the reason its often used as part of a stacker. And with the pain numbing effects, allows you to push harder.
caffiene > jack3d or anything else sold in small tubs. for me at least
I'm sure there is much proof (broscience) that shows A isn't really needed in an ECA stack. Unless you are talking about another stack, ECA is generally where asprin is mentioned.
I suppose it depends on waht you're trying to achieve. It wouldnt be beneficial with regards to weight training. I havent done a sports degree, but I have done my CYQ in sports and exercise fitness, level 1 and level 2.
My thoughts on the matter?
1. Degraded protein synthesis
2. Reduced testosterone
I suppose it could help numb pain.
But with numbing, would come bad posture.
So no, I cant see it helping very much. The cons out weigh the pros
Interestingly, in yeast, different doses of ethanol induce different responses. Over 4%v/v ethanol doses optimally induce heat shock promoters, while 6–8%, induce the same two major changes in integral plasma-membrane protein composition as sublethal heat stress. These changes include reduction in levels of the plasma membrane ATPase protein, and acquisition of the plasma membrane heat-shock protein Hsp30 (Piper et al. 1994). In humans, a beneficial induction of plasma antioxidants is achieved with one drink (5% v/v alcohol) while an increased pro-oxidant state occurs after three drinks from volunteers averaged over 360 minutes. One drink of red wine, beer, or stout provided equivalent increases in plasma antioxidant activity without induction of pro-oxidative stress (*****ett et al. 2004). Thus, at different doses, different physiological responses occur from benefit to harmful response.
Heat-Shock Proteins (HSPs) are involved in growth and development of uscles, however, the exact role that hyperthermia and HSPs play in muscle hypertrophy and muscle repair is unknown. Here, for the first time, we present our findings on the effects of heat shock (HS) in representative cell culture models of cardiac (HL-1), skeletal (C2C12) and smooth (A7R5) muscle cells. We used fully differentiated cells, and exposed them to a mild heat shock protocol (43ºC for 20 min). Cells were returned after HS to their normal culture environment and 24 hrs after HS, we measured cell area. Surprisingly, we discovered that HS induced significant hypertrophy in all three types of muscle cells. To begin deciphering the molecular machinery involved with the hypertrophic response we used a real-time PCR array that included specific genes involved in hypertrophy, cell signaling and transduction, and excitation-contraction coupling. In C2C12, 6 hrs after HS, we found an increase in the expression of Hsp genes and downregulation of IP3R-1, TLR2, MHC6, and Titin. This is the first report to show that HS promotes cardiac, skeletal, and smooth muscle hypertrophy that associates with changes in a small subset of genes.